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16+ results
Field: Metabolism and Genetic Disorders

Disorders of carnitine transport and the carnitine cycle

Verified

Nicola Longo, Cristina Amat Di San Filippo, Marzia Pasquali

Journal: American Journal of Medical Genetics Part C Seminars in Medical Genetics
Year: 2006
Citations: 540

Carnitine plays an essential role in the transfer of long-chain fatty acids across the inner mitochondrial membrane. This transfer requires enzymes and transporters that accumulate carnitine within the cell (OCTN2 carnitine transporter), conjugate it with long chain fatty acids (carnitine palmitoyl ...

Life SciencesBiochemistry, Genetics and Molecular BiologyClinical BiochemistryOpen Access
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Deficiency of Asparagine Synthetase Causes Congenital Microcephaly and a Progressive Form of Encephalopathy

Verified

Elizabeth K. Ruzzo, José‐Mario Capo‐Chichi, Bruria Ben‐Zeev, David Chitayat et al.

Journal: NeuronYear: 2013Citations: 166

We analyzed four families that presented with a similar condition characterized by congenital microcephaly, intellectual disability, progressive cerebral atrophy, and intractable seizures. We show that recessive mutations in the ASNS gene are responsible for this syndrome. Two of the identified miss...

Life SciencesBiochemistry, Genetics and Molecular BiologyBiochemistryOpen Access
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Glutaric acidemia type 1

Verified

Gary L. Hedlund, Nicola Longo, Marzia Pasquali

Journal: American Journal of Medical Genetics Part C Seminars in Medical GeneticsYear: 2006Citations: 129

Glutaric acidemias comprise different disorders resulting in an increased urinary excretion of glutaric acid. Glutaric acidemia type 1 (GA-1) is an autosomal recessive disorder of lysine, hydroxylysine, and tryptophan metabolism caused by deficiency of glutaryl-CoA dehydrogenase. It results in the a...

Life SciencesBiochemistry, Genetics and Molecular BiologyClinical BiochemistryOpen Access
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FETAL LIVER BIOPSY FOR PRENATAL DIAGNOSIS OF ORNITHINE CARBAMYL TRANSFERASE DEFICIENCY

Verified

Charles H. Rodeck, Marcus Pembrey, A. D. Patrick, C Tzannatos et al.

Journal: The LancetYear: 1982Citations: 72
Life SciencesBiochemistry, Genetics and Molecular BiologyClinical Biochemistry
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Increased peripheral blood mitochondrial DNA in type 2 diabetic patients with nephropathy

Verified

Afshan N. Malik, Rojeen Shahni, Muhammad Usman Iqbal

Journal: Diabetes Research and Clinical PracticeYear: 2009Citations: 62

We report that mitochondrial DNA (MtDNA) copy numbers are increased in peripheral blood of patients with diabetic nephropathy (DN). Using qPCR for quantitation, we found a 2-4-fold significant increase (p<0.05) in the mean MtDNA values in DN patients vs. healthy controls and diabetics without nephro...

Life SciencesBiochemistry, Genetics and Molecular BiologyClinical Biochemistry
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Exogenous Oleic Acid and Palmitic Acid Improve Boar Sperm Motility via Enhancing Mitochondrial Β-Oxidation for ATP Generation

Verified

Zhendong Zhu, Rongnan Li, Chengwen Feng, Ruifang Liu et al.

Journal: AnimalsYear: 2020Citations: 58

It takes several hours for mammalian sperm to migrate from the ejaculation or insemination site to the fertilization site in the female reproductive tract in which glucose, amino acids, and fatty acids are regarded as the primary substrates for ATP generation. The present study was designed to inves...

Health SciencesMedicineReproductive MedicineOpen Access
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Newborn screening in southeastern Europe

Verified

Urh Grošelj, Mojca Žerjav Tanšek, Andraž Šmon, Natalija Angelkova et al.

Journal: Molecular Genetics and MetabolismYear: 2014Citations: 57

The aim of our study was to assess the current state of newborn screening (NBS) in the region of southeastern Europe, as an example of a developing region, focusing also on future plans. Responses were obtained from 11 countries. Phenylketonuria screening was not introduced in four of 11 countries, ...

Life SciencesBiochemistry, Genetics and Molecular BiologyClinical Biochemistry
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Bone dysplasia associated with phytanic acid accumulation and deficient plasmalogen synthesis: A peroxisomal entity amenable to plasmapheresis

Verified

Jan Smeıtınk, F. A. Beemer, Marc Espeel, R. A. M. G. Donckerwocke et al.

Journal: Journal of Inherited Metabolic DiseaseYear: 1992Citations: 57
Life SciencesBiochemistry, Genetics and Molecular BiologyMolecular Biology
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Phenylketonuria screening and management in southeastern Europe – survey results from 11 countries

Verified

Mojca Žerjav Tanšek, Urh Grošelj, Natalija Angelkova, D Anton et al.

Journal: Orphanet Journal of Rare DiseasesYear: 2015Citations: 53

BACKGROUND: We aimed to assess the current state of PKU screening and management in the region of southeastern Europe. METHODS: A survey was performed involving all identified professionals responsible for the PKU management in the 11 countries from South-Eastern region of Europe (Albania, Bulgaria,...

Life SciencesBiochemistry, Genetics and Molecular BiologyClinical BiochemistryOpen Access
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Defining the Newborn Blood Spot Screening Reference Interval for TSH: Impact of Ethnicity

Verified

Catherine Peters, Ivan Brooke, Simon Heales, Adeboye Ifederu et al.

Journal: The Journal of Clinical Endocrinology & MetabolismYear: 2016Citations: 47

CONTEXT: There is variability in the congenital hypothyroidism (CH) newborn screening TSH cutoff across the United Kingdom. OBJECTIVE: To determine the influences of year, gender, and ethnicity on screening variability and examine whether there is an optimal operational TSH cutoff. DESIGN AND SETTIN...

Health SciencesMedicineEndocrinology, Diabetes and MetabolismOpen Access
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Treatment of homocystinuria with pyridoxine. A preliminary study.

Verified

N. A. J. Carson, I. J. Carré

Journal: Archives of Disease in ChildhoodYear: 1969Citations: 47

Homocystinuria is the result of an inborn error in the metabolism of the essential sulphur-containing amino acid, methionine (Carson and Neill, 1962; Gerritsen, Vaughn, and Waisman, 1962). There is now good evidence This results in an accumulation of homo- cysteine in the blood, and as this is a 'no...

Health SciencesMedicineRheumatologyOpen Access
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Biochemical findings in common inborn errors of metabolism

Verified

Marzia Pasquali, Gavin Monsen, Leah D’Aurora Richardson, Martha Alston et al.

Journal: American Journal of Medical Genetics Part C Seminars in Medical GeneticsYear: 2006Citations: 38

The application of tandem mass spectrometry (MS/MS) to newborn screening has led to the detection of patients with a wider spectrum of inborn errors of metabolism. A definitive diagnosis can often be established early enough to start treatment before symptoms appear. Here, we review common biochemic...

Life SciencesBiochemistry, Genetics and Molecular BiologyClinical Biochemistry
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Osteopenia and phenylketonuria

Verified

D. J. Carson, L. G. Greeves, L Sweeney, Michael Crone

Journal: Pediatric RadiologyYear: 1990Citations: 36

Trabecular bone mineral content was assessed by quantitative computed tomography in eleven young adults with phenylketonuria who had been treated from early childhood with a diet restricted in natural protein and supplemented with amino acids, minerals and vitamins. There was a significant reduction...

Life SciencesBiochemistry, Genetics and Molecular BiologyClinical Biochemistry
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Age-Specific Cut-off Values of Amino Acids and Acylcarnitines for Diagnosis of Inborn Errors of Metabolism Using Liquid Chromatography Tandem Mass Spectrometry

Verified

Suprovath Kumar Sarker, Md Tarikul Islam, Aparna Biswas, Golam Sarower Bhuyan et al.

Journal: BioMed Research InternationalYear: 2019Citations: 34

Liquid Chromatography tandem mass spectrometry (LC-MS/MS) is used for the diagnosis of more than 30 inborn errors of metabolisms (IEMs). Accurate and reliable diagnosis of IEMs by quantifying amino acids (AAs) and acylcarnitines (ACs) using LC-MS/MS systems depend on the establishment of age-specifi...

Life SciencesBiochemistry, Genetics and Molecular BiologyClinical BiochemistryOpen Access
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A Novel Mutation in the <i>Albumin</i> Gene (R218S) Causing Familial Dysalbuminemic Hyperthyroxinemia in a Family of Bangladeshi Extraction

Verified

Solomon Maximo Greenberg, Alfonso Massimiliano Ferrara, Everton S. Nicholas, Alexandra M. Dumitrescu et al.

Journal: ThyroidYear: 2014Citations: 29

BACKGROUND: Familial dysalbuminemic hyperthyroxinemia (FDH) is a common cause of euthyroid hyperthyroxinemia. Clinical recognition of FDH is crucial for preventing unnecessary therapy in clinically euthyroid patients with abnormal thyroid function tests. Our goal was to identify the cause of abnorma...

Health SciencesMedicineEndocrinology, Diabetes and MetabolismOpen Access
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